Delayed‐onset dystonia associated with corticospinal tract dysfunction
Identifieur interne : 006551 ( Main/Exploration ); précédent : 006550; suivant : 006552Delayed‐onset dystonia associated with corticospinal tract dysfunction
Auteurs : Stewart A. Factor [États-Unis] ; Juan Sanchez-Ramos [États-Unis] ; Weiner [États-Unis]Source :
- Movement Disorders [ 0885-3185 ] ; 1988.
Descripteurs français
- Pascal (Inist)
- Wicri :
- topic : Homme.
English descriptors
- KwdEn :
- Adult, Aged, Cerebral Infarction (complications), Cerebral infarction, Child, Complication, Computerized axial tomography, Corticospinal bundle, Corticospinal tract, Delayed‐onset dystonia, Dystonia, Dystonia (drug therapy), Dystonia (etiology), Dystonia (pathology), Hemidystonia, Hemiplegia (etiology), Human, Humans, Male, Middle Aged, Muscle tonus alteration, Nervous system diseases, Pyramidal Tracts (pathology), Radiodiagnosis, Time interval, Trihexiphenidyl, Trihexyphenidyl (therapeutic use), Unilateral.
- MESH :
- chemical , therapeutic use : Trihexyphenidyl.
- complications : Cerebral Infarction.
- drug therapy : Dystonia.
- etiology : Dystonia, Hemiplegia.
- pathology : Dystonia, Pyramidal Tracts.
- Adult, Aged, Child, Humans, Male, Middle Aged.
Abstract
We report a young man with delayed‐onset dystonia secondary to a cerebral infarction. The infarction was caused by an embolic event at the age of 18 months. Delay to onset of dystonia was 16 years and, prior to the onset of dystonia, the patient had profound corticospinal tract dysfunction manifested by a severe hemiparesis. Computed tomography scan revealed infarction of the lentiform nucleus and precentral gyrus. The profound involvement of the corticospinal tract both clinically and radiologically are features not previously described in symptomatic dystonia and contradict the concept that sparing of the corticospinal tract is an essential feature for the onset of acquired dystonia.
Url:
DOI: 10.1002/mds.870030304
Affiliations:
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Le document en format XML
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Disord.</title><idno type="ISSN">0885-3185</idno><idno type="eISSN">1531-8257</idno><imprint><publisher>Wiley Subscription Services, Inc., A Wiley Company</publisher><pubPlace>Hoboken</pubPlace><date type="published" when="1988">1988</date><biblScope unit="vol">3</biblScope><biblScope unit="issue">3</biblScope><biblScope unit="page" from="201">201</biblScope><biblScope unit="page" to="210">210</biblScope></imprint><idno type="ISSN">0885-3185</idno></series><idno type="istex">42C53C3971FCFCA7919168890C8BDF9E3457E290</idno><idno type="DOI">10.1002/mds.870030304</idno><idno type="ArticleID">MDS870030304</idno></biblStruct></sourceDesc><seriesStmt><idno type="ISSN">0885-3185</idno></seriesStmt></fileDesc><profileDesc><textClass><keywords scheme="KwdEn" xml:lang="en"><term>Adult</term><term>Aged</term><term>Cerebral Infarction (complications)</term><term>Cerebral infarction</term><term>Child</term><term>Complication</term><term>Computerized axial tomography</term><term>Corticospinal bundle</term><term>Corticospinal tract</term><term>Delayed‐onset dystonia</term><term>Dystonia</term><term>Dystonia (drug therapy)</term><term>Dystonia (etiology)</term><term>Dystonia (pathology)</term><term>Hemidystonia</term><term>Hemiplegia (etiology)</term><term>Human</term><term>Humans</term><term>Male</term><term>Middle Aged</term><term>Muscle tonus alteration</term><term>Nervous system diseases</term><term>Pyramidal Tracts (pathology)</term><term>Radiodiagnosis</term><term>Time interval</term><term>Trihexiphenidyl</term><term>Trihexyphenidyl (therapeutic use)</term><term>Unilateral</term></keywords><keywords scheme="MESH" type="chemical" qualifier="therapeutic use" xml:lang="en"><term>Trihexyphenidyl</term></keywords><keywords scheme="MESH" qualifier="complications" xml:lang="en"><term>Cerebral Infarction</term></keywords><keywords scheme="MESH" qualifier="drug therapy" xml:lang="en"><term>Dystonia</term></keywords><keywords scheme="MESH" qualifier="etiology" xml:lang="en"><term>Dystonia</term><term>Hemiplegia</term></keywords><keywords scheme="MESH" qualifier="pathology" xml:lang="en"><term>Dystonia</term><term>Pyramidal Tracts</term></keywords><keywords scheme="MESH" xml:lang="en"><term>Adult</term><term>Aged</term><term>Child</term><term>Humans</term><term>Male</term><term>Middle Aged</term></keywords><keywords scheme="Pascal" xml:lang="fr"><term>Complication</term><term>Dystonie</term><term>Faisceau corticospinal</term><term>Homme</term><term>Intervalle temps</term><term>Radiodiagnostic</term><term>Ramollissement cérébral</term><term>Système nerveux pathologie</term><term>Tomodensitométrie</term><term>Trouble tonus</term><term>Unilatéral</term></keywords><keywords scheme="Wicri" type="topic" xml:lang="fr"><term>Homme</term></keywords></textClass><langUsage><language ident="en">en</language></langUsage></profileDesc></teiHeader><front><div type="abstract" xml:lang="en">We report a young man with delayed‐onset dystonia secondary to a cerebral infarction. The infarction was caused by an embolic event at the age of 18 months. Delay to onset of dystonia was 16 years and, prior to the onset of dystonia, the patient had profound corticospinal tract dysfunction manifested by a severe hemiparesis. Computed tomography scan revealed infarction of the lentiform nucleus and precentral gyrus. The profound involvement of the corticospinal tract both clinically and radiologically are features not previously described in symptomatic dystonia and contradict the concept that sparing of the corticospinal tract is an essential feature for the onset of acquired dystonia.</div></front></TEI><affiliations><list><country><li>États-Unis</li></country><region><li>Floride</li></region></list><tree><country name="États-Unis"><region name="Floride"><name sortKey="Factor, Stewart A" sort="Factor, Stewart A" uniqKey="Factor S" first="Stewart A." last="Factor">Stewart A. Factor</name></region><name sortKey="Sanchez Amos, Juan" sort="Sanchez Amos, Juan" uniqKey="Sanchez Amos J" first="Juan" last="Sanchez-Ramos">Juan Sanchez-Ramos</name><name sortKey="Weiner" sort="Weiner" uniqKey="Weiner" last="Weiner">Weiner</name></country></tree></affiliations></record>Pour manipuler ce document sous Unix (Dilib)
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